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Pervasive Co-Morbidity and Abdominal Herniation: an Outline

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Recurrent Hernia

Abstract

For most of the 20th century, there was unanimity regarding the etiology of herniae. They arose from congenital defects in normal abdominal wall. The ability to resist protrusion was compromised by straining, poor muscular tone, obesity, debility, ascites, or advancing age. Care was to be undertaken solely by surgeons since repair was curative, provided that recurrence from faulty technique, surgical error, or infection was avoided. Keith [1] did state in 1924 that a pathologic change in the connective tissues of the belly might render certain individuals particularly liable to hernia, but his insight was ignored. The purpose of this review is to outline the evidence which has led to our present understanding regarding the role of co-morbidity in the development of herniae and the implications thereof.

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Read, R.C. (2007). Pervasive Co-Morbidity and Abdominal Herniation: an Outline. In: Schumpelick, V., Fitzgibbons, R.J. (eds) Recurrent Hernia. Springer, Berlin, Heidelberg. https://doi.org/10.1007/978-3-540-68988-1_4

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  • DOI: https://doi.org/10.1007/978-3-540-68988-1_4

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