“Beer potomania” in non-beer drinkers: Effect of low dietary solute intake
References (0)
Cited by (98)
Age-Associated Abnormalities of Water Homeostasis
2023, Endocrinology and Metabolism Clinics of North AmericaPolyuria in adults. A diagnostic approach based on pathophysiology
2022, Revista Clinica EspanolaUse of Urine Electrolytes and Urine Osmolality in the Clinical Diagnosis of Fluid, Electrolytes, and Acid-Base Disorders
2021, Kidney International ReportsCitation Excerpt :In contrast, in the group of patients with the syndrome of inappropriate secretion of antidiuretic hormone (SIADH) or the syndrome of inappropriate antidiuresis to include patients with mutations in AVPR2 causing it to be constitutively active, the release of AVP is not caused by low EABV.74,75 Notwithstanding, the degree of decreased EABV in some of the patients who are considered in the first group (e.g., some patients with thiazide-induced hyponatremia, patients with “tea and toast” hyponatremia) does not seem to be large enough to cause the release of AVP.76–79 Reduced EFW excretion in these patients may be caused by a decreased volume of filtrate delivered to the distal nephron as a result of increased reabsorption in the PCT in response to a mild degree of EABV contraction, particularly in elderly patients with reduced glomerular filtration rate, and the presence of other mechanisms for water reabsorption in the distal nephron that are independent of AVP actions.79–82
The Distribution of Plasma Electrolytes
2017, American Journal of the Medical SciencesTen common pitfalls in the evaluation of patients with hyponatremia
2016, European Journal of Internal MedicineCitation Excerpt :This results in a limited amount of solutes to be excreted and leads to decreased water excretion. This phenomenon has been reported in ill-nourished heavy drinkers (beer potomania syndrome) but also in elderly with increased water and low solute intake (for example, tea and toast diet) [19–22]. Psychiatric patients with increased water intake (partially due to drug-associated xerostomia) and impaired renal diluting ability (underlying renal disease, volume depletion, decreased solute intake or drugs affecting water homeostasis such as thiazide diuretics) may exhibit hyponatremia with relatively dilute urine but with Uosm > 100 mosm/kg (usually between 100 and 300 mosm/kg) [6].
Evaluation of polyuria: The roles of solute loading and water diuresis
2016, American Journal of Kidney Diseases