Estrogens in Pregnancy
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Does exercise during pregnancy impact organs or structures of the maternal-fetal interface?
2021, Tissue and CellCitation Excerpt :A coordinated interdependence of maternal, placental and fetal circulation is necessary for oestrogen synthesis from its precursor, cholesterol. The placenta is devoid of the enzyme dehydroepiandrosterone sulphate and 16 alpha-hydroxy-dehydroepiandrosterone sulphate necessary for the synthesis of different kinds of oestrogens (Levitz and Young, 1978). Instead, the placenta benefits from the availability of these enzymes within fetal and maternal circulation (Levitz and Young, 1978).
Passively acquired lupus in the fetus and neonate
2021, Lahita’s Systemic Lupus ErythematosusThe association between maternal lipid profile after birth and offspring risk of autism spectrum disorder
2021, Annals of EpidemiologyCitation Excerpt :This observed inverse association between LDL concentration and ASD may be a result of abnormal maternal lipid regulation in obese gravidas. Under physiologic pregnancy conditions, the placental production of estrogen progressively increases with gestational age [25]. The rise in estrogen suppresses peripheral lipoprotein lipase activities [15].
Functional significance of lymphocytes in pregnancy and lymphocyte immunotherapy in infertility: A comprehensive review and update
2020, International ImmunopharmacologyEssential oils disrupt steroidogenesis in a feto-placental co-culture model
2019, Reproductive ToxicologyCitation Excerpt :Moreover, it has been recently reported that neonicotinoids thiaclopid and thiamethoxam also possesses this non-monotonic effect in H295R cells [31]. During pregnancy, estriol is a unique estrogen that is produced by the placenta from a fetal androgen precursor, 16α-DHEA-sulfate, which is produced by the fetal liver [67,68]. Fetal hepatic 16α-hydroxylation is mediated by the cytochrome P450 enzyme CYP3A7.
Serotonin-estrogen interactions: What can we learn from pregnancy?
2019, BiochimieCitation Excerpt :In the former, which compose the chorionic villi, cells fuse and form an endocrine–active multinucleated syncytium, the syncytiotrophoblast cells while in the latter, cells invade the maternal decidua to regulate blood flow [285–288]. The human villous trophoblasts produce a large quantity of estrogens, as well as 5-HT, making it an interesting model to study 5-HT-estrogen interaction [289–291]. Pregnancy has been known for a long time to be a hyperestrogenic state [289].