ViewpointA multistep molecular mimicry hypothesis for the pathogenesis of rheumatoid arthritis
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2012, Cellular ImmunologyCitation Excerpt :It has been shown that self-tolerance is broken in BALB/c mice during the induction of PGIA, as the arthritic mice exhibit reactivity with self PG [17], as well as with PG-unrelated other antigens such as citrullinated proteins and self IgG [18]. Such reactivities are thought to arise as a result of intra- and intermolecular epitope spreading in either BALB/c mice with PGIA or patients with RA [18,34–37]. It is likely that APCs in the LNs of mice with PGIA are able to present a large repertoire of antigenic peptides with diverse specificities, for many of which cognate T cells may exist in the transferred naïve, as well as in the antigen-experienced populations.
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