Macular edema secondary to occulusion of the retinal veins

https://doi.org/10.1016/0039-6257(84)90228-5Get rights and content

Abstract

Occlusion of the central and branch retinal veins causes macular edema by provoking increased venous pressure which is transmitted to the perifoveal capillaries. Damage to the perifoveal capillary endothelium results in leakage with macular edema. The severity of the retinal findings (i.e., macular hemorrhages, macular edema, etc.) is determined by the location, completeness, duration, and evolution of the obstruction (i.e., recanalization of a thrombus). Spontaneous remission of macular edema secondary to either a partial central or branch occlusion caries a relative good visual prognosis. Chronic macular edema secondary to occlusion of either central or branch veins is associated with a relatively poor visual prognosis. In selected cases, paramacular photocoagulation can reverse chronic macular edema and result in visual improvement.

References (16)

There are more references available in the full text version of this article.

Cited by (62)

  • Therapies for macular edema associated with central retinal vein occlusion: A report by the American Academy of Ophthalmology

    2015, Ophthalmology
    Citation Excerpt :

    Visual acuity loss after CRVO commonly occurs as a result of ME but also may result from macular ischemia or neovascular complications of CRVO (e.g., vitreous hemorrhage, neovascular glaucoma). Because early, smaller prospective studies reported the benefit of laser photocoagulation for ME associated with CRVO,6,7 macular grid-pattern photocoagulation was evaluated in the context of the National Eye Institute (NEI)-sponsored Central Vein Occlusion Study.8 Corticosteroid-based pharmacotherapies are thought to decrease edema by stabilizing vascular permeability, downregulating inflammatory mediators, and indirectly inhibiting the actions of VEGF.9

  • Branch vein occlusion

    2012, Retina Fifth Edition
View all citing articles on Scopus
View full text